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human aortic smooth muscle cells smcs  (ATCC)


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    ATCC human aortic smooth muscle cells smcs
    Human Aortic Smooth Muscle Cells Smcs, supplied by ATCC, used in various techniques. Bioz Stars score: 96/100, based on 1111 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/human+aortic+smc/T%2FG+HA-VSMC/pmc13019204-95-4-12
    Average 96 stars, based on 1111 article reviews
    human aortic smooth muscle cells smcs - by Bioz Stars, 2026-09
    96/100 stars

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    Related Articles

    other:

    Article Title: A signaling cascade mediated by ceramide, src and PDGFRβ coordinates the activation of the redox-sensitive neutral sphingomyelinase-2 and sphingosine kinase-1.
    Article Snippet: Article history: Received 27 December 2012 Received in revised form 8 March 2013 Accepted 30 April 2013 Available online 4 May 2013

    Transfection:

    Article Title: Role of Sphingosine-1-Phosphate in Transplant Vasculopathy Evoked by Anti-HLA Antibody.
    Article Snippet: Transplant vasculopathy (TV) represents the main cause of late graft failure and limits the long-term success of organ transplantation.. Cellular and humoral immune responses contribute to the pathogenesis of the concentric and diffuse intimal hyperplasia of arteries of the grafted organ.. We recently reported that the mitogenic signaling, evoked in human vascular smoothmuscle cells (hmSMC) by the anti-HLA class I monoclonal antibody W6/32, implicates neutral sphingomyelinase-2, suggesting a role for sphingolipids in intimal hyperplasia of TV.

    Activity Assay:

    Article Title: Anticoagulant compounds and methods and devices for their use
    Article Snippet: .. Anti-proliferative activity of Apixaban, Argatroban, and Rapamycin was tested in Human Aortic SMC (HAoSMC, ATCC, PCS-100-012). .. Anti-proliferative activity of Apixaban, Argatroban, and Rapamycin was tested in Human Aortic SMC (HAoSMC, ATCC, PCS-100-012).

    Article Title: Anticoagulant compounds and methods and devices for their use
    Article Snippet: .. Anti-proliferative activity of Apixaban, Argatroban, and Rapamycin was tested in Human Aortic SMC (HAoSMC, ATCC, PCS-100-012). ..



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    human aortic smcs  (ATCC)
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    A. Outline of the experiment. Human <t>aortic</t> <t>SMCs</t> were exposed to static conditions (24 h), mechanical stretch (10%, 6h) or TNF stimulation (10 ng/mL, 24h). Uniform Manifold Approximation and Projection (UMAP) visualization of the phenotypic diversity of SMCs after static culture, stretch and TNF stimulation. Each dot represents a single cell. Nine cell clusters were identified in human aortic SMCs (0.3 resolution) as follows: C0 (fibromyocyte SMCs), C1 (inflammatory SMCs), C2 (proliferative M-phase), C3 (intermediate SMCs), C4 (proliferative S-phase), C5 (proliferative M-S-phase), C6 (contractile SMCs), C7 (apoptotic SMCs), C8 (endothelial-like SMCs). B. Dot plot showing the expression of three genes defining each cell cluster shown in A. Color scale indicates log2 expression, and the size represents the percentage of cells. C. Integration of publicly available human coronary and carotid atherosclerotic plaques scRNA-seq datasets – with human aortic SMCs from this study. This analysis revealed 7 mesenchymal cell clusters (pC0 to pC6). The p stands for plaque. Analysis of human aortic SMCs in culture are shown in the background (light grey cluster). D. Human aortic SMCs (C0 to C8) are shown in front and human atherosclerotic cell populations are shown in the background (light grey clusters).
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    primary human aortic smcs  (ATCC)
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    A. Outline of the experiment. Human <t>aortic</t> <t>SMCs</t> were exposed to static conditions (24 h), mechanical stretch (10%, 6h) or TNF stimulation (10 ng/mL, 24h). Uniform Manifold Approximation and Projection (UMAP) visualization of the phenotypic diversity of SMCs after static culture, stretch and TNF stimulation. Each dot represents a single cell. Nine cell clusters were identified in human aortic SMCs (0.3 resolution) as follows: C0 (fibromyocyte SMCs), C1 (inflammatory SMCs), C2 (proliferative M-phase), C3 (intermediate SMCs), C4 (proliferative S-phase), C5 (proliferative M-S-phase), C6 (contractile SMCs), C7 (apoptotic SMCs), C8 (endothelial-like SMCs). B. Dot plot showing the expression of three genes defining each cell cluster shown in A. Color scale indicates log2 expression, and the size represents the percentage of cells. C. Integration of publicly available human coronary and carotid atherosclerotic plaques scRNA-seq datasets – with human aortic SMCs from this study. This analysis revealed 7 mesenchymal cell clusters (pC0 to pC6). The p stands for plaque. Analysis of human aortic SMCs in culture are shown in the background (light grey cluster). D. Human aortic SMCs (C0 to C8) are shown in front and human atherosclerotic cell populations are shown in the background (light grey clusters).
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    A. Outline of the experiment. Human aortic SMCs were exposed to static conditions (24 h), mechanical stretch (10%, 6h) or TNF stimulation (10 ng/mL, 24h). Uniform Manifold Approximation and Projection (UMAP) visualization of the phenotypic diversity of SMCs after static culture, stretch and TNF stimulation. Each dot represents a single cell. Nine cell clusters were identified in human aortic SMCs (0.3 resolution) as follows: C0 (fibromyocyte SMCs), C1 (inflammatory SMCs), C2 (proliferative M-phase), C3 (intermediate SMCs), C4 (proliferative S-phase), C5 (proliferative M-S-phase), C6 (contractile SMCs), C7 (apoptotic SMCs), C8 (endothelial-like SMCs). B. Dot plot showing the expression of three genes defining each cell cluster shown in A. Color scale indicates log2 expression, and the size represents the percentage of cells. C. Integration of publicly available human coronary and carotid atherosclerotic plaques scRNA-seq datasets – with human aortic SMCs from this study. This analysis revealed 7 mesenchymal cell clusters (pC0 to pC6). The p stands for plaque. Analysis of human aortic SMCs in culture are shown in the background (light grey cluster). D. Human aortic SMCs (C0 to C8) are shown in front and human atherosclerotic cell populations are shown in the background (light grey clusters).

    Journal: bioRxiv

    Article Title: Mechanical stretch regulates inflammatory signaling in human smooth muscle cells

    doi: 10.1101/2025.10.29.685276

    Figure Lengend Snippet: A. Outline of the experiment. Human aortic SMCs were exposed to static conditions (24 h), mechanical stretch (10%, 6h) or TNF stimulation (10 ng/mL, 24h). Uniform Manifold Approximation and Projection (UMAP) visualization of the phenotypic diversity of SMCs after static culture, stretch and TNF stimulation. Each dot represents a single cell. Nine cell clusters were identified in human aortic SMCs (0.3 resolution) as follows: C0 (fibromyocyte SMCs), C1 (inflammatory SMCs), C2 (proliferative M-phase), C3 (intermediate SMCs), C4 (proliferative S-phase), C5 (proliferative M-S-phase), C6 (contractile SMCs), C7 (apoptotic SMCs), C8 (endothelial-like SMCs). B. Dot plot showing the expression of three genes defining each cell cluster shown in A. Color scale indicates log2 expression, and the size represents the percentage of cells. C. Integration of publicly available human coronary and carotid atherosclerotic plaques scRNA-seq datasets – with human aortic SMCs from this study. This analysis revealed 7 mesenchymal cell clusters (pC0 to pC6). The p stands for plaque. Analysis of human aortic SMCs in culture are shown in the background (light grey cluster). D. Human aortic SMCs (C0 to C8) are shown in front and human atherosclerotic cell populations are shown in the background (light grey clusters).

    Article Snippet: Human aortic SMCs (ATCC, PCS-100-012, male donor) were authenticated by ATCC and tested negative for mycoplasma contamination prior to experiments.

    Techniques: Expressing